title: Defective Wnt Signaling: A Potential Contributor to Cardiometabolic Disease?
authors.affiliation: Division of Clinical Pharmacology and Vanderbilt Autonomic Dysfunction Center, Vanderbilt University School of Medicine, Nashville, TN.
authors.forename: Amy C
authors.initials: AC
authors.lastname: Arnold
authors.affiliation: Division of Clinical Pharmacology and Vanderbilt Autonomic Dysfunction Center, Vanderbilt University School of Medicine, Nashville, TN david.robertson@vanderbilt.edu.
authors.forename: David
authors.initials: D
authors.lastname: Robertson
mesh_terms: D000818:Animals; D001794:Blood Pressure / Q000502:physiology*; D038362:Glycogen Synthase Kinase 3 / Q000378:metabolism*; D007328:Insulin / Q000502:physiology*; D055504:Insulin Receptor Substrate Proteins / Q000378:metabolism*; D008297:Male; D017552:Solitary Nucleus / Q000502:physiology*; D051153:Wnt Proteins / Q000378:metabolism*
chemical_list: D007328:Insulin; D055504:Insulin Receptor Substrate Proteins; D051153:Wnt Proteins; D038362:Glycogen Synthase Kinase 3