title: [Systemic factors of acute pancreatitis advance].
abstract: In experimental studies the sequence of vitals series lesion is established at various forms of acute experimental pancreatitis (edematous pancreatitis, a pancreatonecrosis). It is shown that one of the first in pathological process involves an intestine, then - a liver, further - kidneys. It is established that at acute pancreatitis the basis of a target organs lesion is membranodestruktivny process due to excess activity of lipids peroxidation processes and phospholipase systems which are registered not only in a blood plasma, but also in tissue structures of the studied organs - an intestine, a liver, kidneys (systemic distress damages of oxidative and phospholipase induced character) acts.
authors.forename: G V
authors.initials: GV
authors.lastname: Poryadin
authors.forename: A P
authors.initials: AP
authors.lastname: Vlasov
authors.forename: S G
authors.initials: SG
authors.lastname: Anasskin
authors.forename: T I
authors.initials: TI
authors.lastname: Vlasova
authors.forename: I V
authors.initials: IV
authors.lastname: Potynova
authors.forename: S A
authors.initials: SA
authors.lastname: Turigina
mesh_terms: D000208:Acute Disease; D000818:Animals; D004285:Dogs; D005260:Female; D007413:Intestinal Mucosa / Q000378:metabolism; D007422:Intestines / Q000473:pathology / Q000503:physiopathology; D007668:Kidney / Q000378:metabolism / Q000473:pathology / Q000503:physiopathology; D015227:Lipid Peroxidation; D008099:Liver / Q000378:metabolism / Q000473:pathology / Q000503:physiopathology; D008297:Male; D010195:Pancreatitis / Q000378:metabolism / Q000473:pathology / Q000503:physiopathology