title: The PCL1-p53 axis promotes cellular quiescence.
authors.affiliation: a The Center for Epigenetics Research, Memorial Sloan Kettering Cancer Center , New York , NY , USA.|b Cancer Biology and Genetics Program, Memorial Sloan Kettering Cancer Center , New York , NY , USA.
authors.forename: Gerard L
authors.initials: GL
authors.lastname: Brien
authors.affiliation: c The Smurfit Institute of Genetics, Trinity College Dublin , Dublin 2 , Ireland.
authors.forename: Adrian P
authors.initials: AP
authors.lastname: Bracken
mesh_terms: D059447:Cell Cycle Checkpoints; D004268:DNA-Binding Proteins / Q000737:chemistry / Q000378:metabolism*; D006801:Humans; D014411:Neoplastic Stem Cells / Q000166:cytology / Q000378:metabolism; D063146:Polycomb-Group Proteins / Q000737:chemistry / Q000378:metabolism*; D017434:Protein Structure, Tertiary; D016159:Tumor Suppressor Protein p53 / Q000737:chemistry / Q000378:metabolism*
chemical_list: D004268:DNA-Binding Proteins; C112309:PHF1 protein, human; D063146:Polycomb-Group Proteins; D016159:Tumor Suppressor Protein p53
keywords: CDKN1A; PCL1; Polycomb-like 1; cancer stem cell; cellular quiescence; chemotherapy; p53