title: Reply: Parsing the differences in affected with LHON: genetic versus environmental triggers of disease conversion.
authors.affiliation: 1 Wellcome Trust Centre for Mitochondrial Research, Institute of Genetic Medicine, Newcastle University, Central Parkway, Newcastle upon Tyne, NE1 3BZ, UK 2 Departments of Neurology and Ophthalmology, Royal Victoria Infirmary, Newcastle upon Tyne, NE1 4LP, UK.
authors.forename: Patrick
authors.initials: P
authors.lastname: Yu-Wai-Man
authors.affiliation: 1 Wellcome Trust Centre for Mitochondrial Research, Institute of Genetic Medicine, Newcastle University, Central Parkway, Newcastle upon Tyne, NE1 3BZ, UK.
authors.forename: Gavin
authors.initials: G
authors.lastname: Hudson
authors.affiliation: 1 Wellcome Trust Centre for Mitochondrial Research, Institute of Genetic Medicine, Newcastle University, Central Parkway, Newcastle upon Tyne, NE1 3BZ, UK 4 German Center for Neurodegenerative Diseases (DZNE), München, Germany 5 Munich Cluster for Systems Neurology (SyNergy), München, Germany.
authors.forename: Thomas
authors.initials: T
authors.lastname: Klopstock
authors.affiliation: 1 Wellcome Trust Centre for Mitochondrial Research, Institute of Genetic Medicine, Newcastle University, Central Parkway, Newcastle upon Tyne, NE1 3BZ, UK 6 Medical Research Council Mitochondrial Biology Unit, Cambridge, UK 7 Department of Clinical Neurosciences, School of Clinical Medicine, University of Cambridge, UK pfc25@medschl.cam.ac.uk.
authors.forename: Patrick F
authors.initials: PF
authors.lastname: Chinnery
mesh_terms: D000818:Animals; D006801:Humans; D029241:Optic Atrophy, Autosomal Dominant / Q000503:physiopathology* / Q000628:therapy*; D029242:Optic Atrophy, Hereditary, Leber / Q000503:physiopathology* / Q000628:therapy*