title: Editorial: Proton pathway paradox: Hv1 H+ channel sustains neutrophil Nox2 activity, yet suppresses HOCl formation.
authors.affiliation: Institute for Integration of Medicine and Science and Department of Medicine, University of Texas Health Science Center, San Antonio, Texas, USA; and South Texas Veterans Healthcare System, San Antonio, Texas, USA clarkra@uthscsa.edu.
authors.forename: Robert A
authors.initials: RA
authors.lastname: Clark
mesh_terms: D000818:Animals; D003594:Cytoplasmic Granules / Q000378:metabolism*; D005260:Female; D007473:Ion Channels / Q000378:metabolism*; D009504:Neutrophils / Q000378:metabolism*
chemical_list: D007473:Ion Channels
keywords: NADPH oxidase; exocytosis; myeloperoxidase; respiratory burst