title: H. pylori's BabA Embraces Change.
abstract: Long before the cause was discovered, peptic ulcers were known to occur preferentially in individuals of certain blood groups. In this issue of Cell Host & Microbe, Moonens et al. (2016) perform structural analyses of the Helicobacter pylori adhesin BabA to determine how the bacteria discriminately bind different blood group glycans.
authors.affiliation: Institute of Molecular Biology, University of Oregon, Eugene, OR 97403, USA.
authors.forename: Emily Goers
authors.initials: EG
authors.lastname: Sweeney
authors.affiliation: Institute of Molecular Biology, University of Oregon, Eugene, OR 97403, USA. Electronic address: guillemin@molbio.uoregon.edu.
authors.forename: Karen
authors.initials: K
authors.lastname: Guillemin
mesh_terms: D018829:Adhesins, Bacterial / Q000378:metabolism; D016481:Helicobacter Infections / Q000382:microbiology*; D016480:Helicobacter pylori / Q000378:metabolism*; D006801:Humans
chemical_list: D018829:Adhesins, Bacterial