title: BCR engagement in CLL: when translation goes wrong.
abstract: In this issue of Blood, Yeomans et al identify MYC as an important target for translational regulation in chronic lymphocytic leukemia (CLL) cells after B-cell receptor (BCR) stimulation and show that current therapies suppress this induction.
authors.affiliation: LUXEMBOURG INSTITUTE OF HEALTH.
authors.forename: Jerome
authors.initials: J
authors.lastname: Paggetti
authors.affiliation: LUXEMBOURG INSTITUTE OF HEALTH.
authors.forename: Etienne
authors.initials: E
authors.lastname: Moussay
mesh_terms: D015973:Gene Expression Regulation, Leukemic; D006801:Humans; D015451:Leukemia, Lymphocytic, Chronic, B-Cell / Q000235:genetics*; D016271:Proto-Oncogene Proteins c-myc / Q000235:genetics*; D012333:RNA, Messenger / Q000235:genetics*; D011947:Receptors, Antigen, B-Cell / Q000276:immunology*
chemical_list: D016271:Proto-Oncogene Proteins c-myc; D012333:RNA, Messenger; D011947:Receptors, Antigen, B-Cell