title: Children's Blood Lead Seasonality in Flint, Michigan (USA), and Soil-Sourced Lead Hazard Risks.
abstract: In Flint; MI; USA; a public health crisis resulted from the switching of the water supply from Lake Huron to a more corrosive source from the Flint River in April 2014; which caused lead to leach from water lines. Between 2010 and 2015; Flint area children's average blood lead patterns display consistent peaks in the third quarter of the year. The third quarter blood lead peaks displayed a declining trend between 2010 and 2013; then rose abruptly between the third quarters of 2013 from 3.6% blood lead levels ≥5 µg/dL to a peak of about 7% in the third quarter of 2014; an increase of approximately 50%. The percentage of blood lead level ≥5 µg/dL in the first quarter of 2015 then dropped to 2.3%; which was the same percentage as the first quarter of 2014 (prior to the Flint River water source change). The Flint quarterly blood lead level peak then rose to about 6% blood lead levels ≥ 5 µg/dL in the third quarter of 2015; and then declined to about 2.5% in the fourth quarter of 2015. Soil lead data collected by Edible Flint food collaborative reveal generally higher soil lead values in the metropolitan center for Flint; with lower values in the outskirts of the city. The questions that are not being asked is why did children's blood lead levels display a seasonal blood lead pattern before the introduction of the new water supply in Flint; and what are the implications of these seasonal blood lead patterns? Based upon previous findings in Detroit and other North American cities we infer that resuspension to the air of lead in the form of dust from lead contaminated soils in Flint appears to be a persistent contribution to lead exposure of Flint children even before the change in the water supply from Lake Huron to the Flint River.
authors.affiliation: Centre for Environmental Sustainability and Remediation (EnSuRe), School of Science, Royal Melbourne Institute of Technology (RMIT) University, PO Box 71, Bundoora, Victoria 3083, Australia. mark.laidlaw@rmit.edu.au.
authors.forename: Mark A S
authors.initials: MA
authors.lastname: Laidlaw
authors.affiliation: Department of Earth Sciences and Center for Urban Health, Indiana University-Purdue University Indianapolis (IUPUI), Indianapolis, IN 46202, USA. gfilippe@iupui.edu.
authors.forename: Gabriel M
authors.initials: GM
authors.lastname: Filippelli
authors.affiliation: Department of Family Medicine, College of Human Medicine, Michigan State University, Flint, MI 48502, USA. sadlerr@msu.edu.
authors.forename: Richard C
authors.initials: RC
authors.lastname: Sadler
authors.affiliation: Department of Pharmacology, Tulane University School of Medicine, New Orleans, LA 70112, USA. cgonza3@tulane.edu.
authors.forename: Christopher R
authors.initials: CR
authors.lastname: Gonzales
authors.affiliation: Centre for Environmental Sustainability and Remediation (EnSuRe), School of Science, Royal Melbourne Institute of Technology (RMIT) University, PO Box 71, Bundoora, Victoria 3083, Australia. andy.ball@rmit.edu.au.
authors.forename: Andrew S
authors.initials: AS
authors.lastname: Ball
authors.affiliation: Department of Pharmacology, Tulane University School of Medicine, New Orleans, LA 70112, USA. hmielke@tulane.edu.
authors.forename: Howard W
authors.initials: HW
authors.lastname: Mielke
mesh_terms: D002648:Child; D002947:Cities; D004391:Dust / Q000032:analysis*; D004781:Environmental Exposure / Q000032:analysis; D004787:Environmental Pollution; D006801:Humans; D007854:Lead / Q000097:blood*; D007855:Lead Poisoning; D008824:Michigan; D011634:Public Health; D012306:Risk; D012621:Seasons; D012987:Soil / Q000737:chemistry*; D012989:Soil Pollutants / Q000097:blood*; D014881:Water Supply
chemical_list: D004391:Dust; D012987:Soil; D012989:Soil Pollutants; D007854:Lead
keywords: Flint; blood lead; lead exposure; lead poisoning; seasonality; soil; water