title: Case report: Unilateral conduction hearing loss due to central venous occlusion.
abstract: Central venous stenosis is a well-known complication in patients with vascular access for hemodialysis. We report two cases involving patients on hemodialysis with arteriovenous fistulas who developed reversible unilateral conductive hearing loss secondary to critical stenosis of central veins draining the arteriovenous dialysis access. A proposed mechanism for the patients' reversible unilateral hearing loss is pterygoid venous plexus congestion leading to decreased Eustachian tube patency. Endovascular therapy was conducted to treat the stenosis and the hearing loss of both patients was returned to near normal after successful central venous angioplasty.
authors.affiliation: Department of Internal Medicine, University of Nevada School of Medicine, Las Vegas, Nevada - USA.
authors.forename: Phillip
authors.initials: P
authors.lastname: Ribeiro
authors.affiliation: Department of Internal Medicine, University of Nevada School of Medicine, Las Vegas, Nevada - USA.
authors.forename: Swetal
authors.initials: S
authors.lastname: Patel
authors.affiliation: Kidney Specialists of Nevada, Las Vegas, Nevada - USA.|Vascular Access Centers of Southern Nevada, Las Vegas, Nevada - USA.
authors.forename: Rizwan A
authors.initials: RA
authors.lastname: Qazi
mesh_terms: D000368:Aged; D000800:Angioplasty, Balloon; D001166:Arteriovenous Shunt, Surgical / Q000009:adverse effects*; D001844:Bone Conduction; D003251:Constriction, Pathologic; D005064:Eustachian Tube / Q000503:physiopathology; D005260:Female; D006314:Hearing Loss, Conductive / Q000175:diagnosis / Q000209:etiology* / Q000503:physiopathology; D046088:Hearing Loss, Unilateral / Q000175:diagnosis / Q000209:etiology* / Q000503:physiopathology; D006801:Humans; D007676:Kidney Failure, Chronic / Q000175:diagnosis / Q000628:therapy*; D008297:Male; D020127:Recovery of Function; D006435:Renal Dialysis; D016896:Treatment Outcome; D014652:Vascular Diseases / Q000000981:diagnostic imaging / Q000209:etiology* / Q000628:therapy