title: More trouble with FGF23: a novel role in systemic immunosuppression.
abstract: A recent study describes impaired neutrophil function as another adverse effect of the excessive levels of FGF23 found in the circulation of patients with chronic kidney disease. These findings shed light on the mechanisms by which chronic kidney disease leads to systemic immunosuppression.
authors.affiliation: Institute of Experimental Immunology, University of Bonn, Bonn, Germany.
authors.forename: Zeinab
authors.initials: Z
authors.lastname: Abdullah
authors.affiliation: Institute of Experimental Immunology, University of Bonn, Bonn, Germany. Electronic address: ckurts@uni-bonn.de.
authors.forename: Christian
authors.initials: C
authors.lastname: Kurts
mesh_terms: D000089703:Fibroblast Growth Factor-23; D005346:Fibroblast Growth Factors; D006801:Humans; D051436:Renal Insufficiency, Chronic
chemical_list: C000717427:FGF23 protein, human; D005346:Fibroblast Growth Factors; D000089703:Fibroblast Growth Factor-23