title: Comment on Goldsworthy et al. Haploinsufficiency of the Insulin Receptor in the Presence of a Splice-Site Mutation in Ppp2r2a Results in a Novel Digenic Mouse Model of Type 2 Diabetes. Diabetes 2016;65:1434-1446.
authors.affiliation: Translational Oncology Division, Oncohealth Institute, Instituto de Investigación Sanitaria de la Fundación Jiménez Díaz, University Hospital "Fundacion Jimenez Diaz," Autonomous University of Madrid, Madrid, Spain ion.cristobal@fjd.es jgfoncillas@fjd.es.
authors.forename: Ion
authors.initials: I
authors.lastname: Cristóbal
authors.affiliation: Pathology Department, University Hospital "Fundacion Jimenez Diaz," Autonomous University of Madrid, Madrid, Spain.
authors.forename: Juan
authors.initials: J
authors.lastname: Madoz-Gúrpide
authors.affiliation: Pathology Department, University Hospital "Fundacion Jimenez Diaz," Autonomous University of Madrid, Madrid, Spain.
authors.forename: Federico
authors.initials: F
authors.lastname: Rojo
authors.affiliation: Translational Oncology Division, Oncohealth Institute, Instituto de Investigación Sanitaria de la Fundación Jiménez Díaz, University Hospital "Fundacion Jimenez Diaz," Autonomous University of Madrid, Madrid, Spain ion.cristobal@fjd.es jgfoncillas@fjd.es.
authors.forename: Jesús
authors.initials: J
authors.lastname: García-Foncillas
mesh_terms: D000818:Animals; D003924:Diabetes Mellitus, Type 2 / Q000235:genetics*; D057895:Haploinsufficiency; D006801:Humans; D007328:Insulin / Q000235:genetics; D051379:Mice; D009154:Mutation; D011972:Receptor, Insulin / Q000235:genetics*
chemical_list: D007328:Insulin; D011972:Receptor, Insulin