title: Galectin-3, Cardiac Function, and Fibrosis.
abstract: This Correspondence relates to the article by Frunza et al (Myocardial Galectin-3 Expression Is Associated with Remodeling of the Pressure-Overloaded Heart and May Delay the Hypertrophic Response without Affecting Survival, Dysfunction, and Cardiac Fibrosis. Am J Pathol 2016, 186:1114-1127).
authors.affiliation: Department of Cardiology, University Medical Center Groningen, University of Groningen, RB Groningen, the Netherlands.
authors.forename: Wouter C
authors.initials: WC
authors.lastname: Meijers
authors.affiliation: Cardiovascular Translational Research, Sanataria de Navarra Research Institute, Pamplona, Spain.
authors.forename: Natalia
authors.initials: N
authors.lastname: López-Andrés
authors.affiliation: Department of Cardiology, University Medical Center Groningen, University of Groningen, RB Groningen, the Netherlands. Electronic address: r.a.de.boer@umcg.nl.
authors.forename: Rudolf A
authors.initials: RA
authors.lastname: de Boer
mesh_terms: D005355:Fibrosis; D037502:Galectin 3; D006321:Heart; D006801:Humans; D009206:Myocardium; D020257:Ventricular Remodeling
chemical_list: D037502:Galectin 3