Platelet derived FasL contributes to apoptosis in stroke.
Peter
P
Kraft
Rebecca
R
Schleicher
Marcus
M
Olbrich
Michael K
MK
Schuhmann
Kelly Daryll
KD
Blanz
Frederic
F
Emschermann
Sonja
S
Ebenhoech
Christopher
C
Starz
Ingo
I
Hilgendorf
Sven G
SG
Meuth
Frank
F
Edlich
Christoph Kleinschnitz, MD, Department of Neurology, University Clinic Essen, Hufelandstr 55, 45147 Essen, Germany, Tel.: +49 201 723 6302, Fax: +49 201 723 5655, E-mail: neurologie@uk-essen.de.
Christoph
C
Kleinschnitz
Harald F. Langer, MD, Section for Cardioimmunology, Eberhard Karls University, Otfried-Mueller-Straße 10, 72076 Tuebingen, Germany, Tel.: +49 7071 2982887, Fax: +49 7071 294474, E-mail: harald.langer@med.uni-tuebingen.de.
Harald F
HF
Langer
D000818:Animals; D017209:Apoptosis; D001792:Blood Platelets / Q000378:metabolism*; D053222:Fas Ligand Protein / Q000378:metabolism*; D006801:Humans; D007249:Inflammation; D019169:Jurkat Cells; D051379:Mice; D008810:Mice, Inbred C57BL; D015539:Platelet Activation; D020521:Stroke / Q000378:metabolism / Q000503:physiopathology*; D013927:Thrombosis / Q000378:metabolism
C505661:FASLG protein, human; D053222:Fas Ligand Protein; C505658:Fasl protein, mouse