title: Hepatitis B virus X protein crosses out Smc5/6 complex to maintain covalently closed circular DNA transcription.
authors.affiliation: Department of Microbiology and Immunology, Indiana University School of Medicine, Indianapolis, IN.
authors.forename: Bidisha
authors.initials: B
authors.lastname: Mitra
authors.affiliation: Department of Microbiology and Immunology, Indiana University School of Medicine, Indianapolis, IN.
authors.forename: Haitao
authors.initials: H
authors.lastname: Guo
mesh_terms: D018797:Cell Cycle Proteins / Q000378:metabolism*; D002868:Chromosomal Proteins, Non-Histone / Q000378:metabolism*; D004270:DNA, Circular / Q000378:metabolism*; D054884:Host-Pathogen Interactions; D006801:Humans; D015534:Trans-Activators / Q000502:physiology*; D014158:Transcription, Genetic; D054334:Viral Regulatory and Accessory Proteins; D014779:Virus Replication
chemical_list: D018797:Cell Cycle Proteins; D002868:Chromosomal Proteins, Non-Histone; D004270:DNA, Circular; C432931:SMC5 protein, human; C432932:SMC6 protein, human; D015534:Trans-Activators; D054334:Viral Regulatory and Accessory Proteins; C057628:hepatitis B virus X protein