title: Cis-eQTLs regulate reduced LST1 gene and NCR3 gene expression and contribute to increased autoimmune disease risk.
authors.affiliation: School of Life Science and Technology, Harbin Institute of Technology, Harbin 150001, China.
authors.forename: Guiyou
authors.initials: G
authors.lastname: Liu
authors.affiliation: School of Life Science and Technology, Harbin Institute of Technology, Harbin 150001, China.
authors.forename: Yang
authors.initials: Y
authors.lastname: Hu
authors.affiliation: Department of Mathematics, Harbin Institute of Technology, Harbin 150001, China.
authors.forename: Shuilin
authors.initials: S
authors.lastname: Jin
authors.affiliation: Department of Neurology and Tianjin Neurological Institute, Tianjin Medical University General Hospital, Tianjin 300052, China.
authors.forename: Fang
authors.initials: F
authors.lastname: Zhang
authors.affiliation: School of Life Science and Technology, Harbin Institute of Technology, Harbin 150001, China; hjw@tmu.edu.cn qhjiang@hit.edu.cn.
authors.forename: Qinghua
authors.initials: Q
authors.lastname: Jiang
authors.affiliation: Department of Neurology and Tianjin Neurological Institute, Tianjin Medical University General Hospital, Tianjin 300052, China hjw@tmu.edu.cn qhjiang@hit.edu.cn.
authors.forename: Junwei
authors.initials: J
authors.lastname: Hao
mesh_terms: D001327:Autoimmune Diseases / Q000235:genetics; D015870:Gene Expression; D005786:Gene Expression Regulation; D055106:Genome-Wide Association Study; D006801:Humans; D020641:Polymorphism, Single Nucleotide; D040641:Quantitative Trait Loci