title: Hyperuricemia: A causal player or a bystander linking inflammatory signaling and atrial fibrillation?
authors.affiliation: Cardiovascular Research Institute; Department of Molecular Physiology & Biophysics, Baylor College of Medicine, Houston, TX, USA. Electronic address: nal@bcm.edu.
authors.forename: Na
authors.initials: N
authors.lastname: Li
authors.affiliation: Institute of Pharmacology, West German Heart and Vascular Center, University Duisburg-Essen, Essen, Germany. Electronic address: dobromir.dobrev@uk-essen.de.
authors.forename: Dobromir
authors.initials: D
authors.lastname: Dobrev
mesh_terms: D001281:Atrial Fibrillation / Q000209:etiology* / Q000378:metabolism; D006801:Humans; D033461:Hyperuricemia / Q000150:complications* / Q000378:metabolism; D058847:Inflammasomes / Q000378:metabolism*; D000071199:NLR Family, Pyrin Domain-Containing 3 Protein / Q000378:metabolism; D012307:Risk Factors; D015398:Signal Transduction; D014527:Uric Acid / Q000097:blood*
chemical_list: D058847:Inflammasomes; D000071199:NLR Family, Pyrin Domain-Containing 3 Protein; C453881:NLRP3 protein, human; D014527:Uric Acid