title: The TBK1-OPTN Axis Mediates Crosstalk Between Mitophagy and the Innate Immune Response: A Potential Therapeutic Target for Neurodegenerative Diseases.
authors.affiliation: Department of Neurosurgery, First Affiliated Hospital, University of South China, Hengyang, 421001, China.
authors.forename: Lu
authors.initials: L
authors.lastname: He
authors.affiliation: Institute of Pharmacy and Pharmacology, Hunan Province Cooperative Innovation Center for Molecular Target New Drug StudyUniversity of South China, Hengyang, 421001, China.
authors.forename: Linxi
authors.initials: L
authors.lastname: Chen
authors.affiliation: Department of Neurosurgery, First Affiliated Hospital, University of South China, Hengyang, 421001, China. llanfang6@126.com.|Institute of Pharmacy and Pharmacology, Hunan Province Cooperative Innovation Center for Molecular Target New Drug StudyUniversity of South China, Hengyang, 421001, China. llanfang6@126.com.
authors.forename: Lanfang
authors.initials: L
authors.lastname: Li
mesh_terms: D001343:Autophagy; D006801:Humans; D007113:Immunity, Innate; D008928:Mitochondria; D063306:Mitophagy; D019636:Neurodegenerative Diseases; D010766:Phosphorylation; D017346:Protein Serine-Threonine Kinases
chemical_list: D017346:Protein Serine-Threonine Kinases; C403191:TBK1 protein, human