title: HIV: Persistence through division.
abstract: A long-lived latent reservoir for HIV-1 persists in CD4+T cells despite antiretroviral therapy and is the major barrier to cure. In this issue of JEM, Hosmane et al. show that T cell proliferation could explain the long-term persistence of this reservoir.
authors.affiliation: Laboratory of Molecular Immunology, The Rockefeller University, New York, NY 10065 and Howard Hughes Medical Institute, Chevy Chase, MD 20815.
authors.forename: Lillian B
authors.initials: LB
authors.lastname: Cohn
authors.affiliation: Laboratory of Molecular Immunology, The Rockefeller University, New York, NY 10065 and Howard Hughes Medical Institute, Chevy Chase, MD 20815.
authors.forename: Michel C
authors.identifier: 0000-0003-0592-8564
authors.initials: MC
authors.lastname: Nussenzweig
mesh_terms: D015496:CD4-Positive T-Lymphocytes / Q000821:virology*; D002455:Cell Division; D015497:HIV-1 / Q000502:physiology*; D006801:Humans; D008213:Lymphocyte Activation; D016662:Virus Integration