RNA-seq of Actinidia chinensis: fruit
Botryosphaeria dothidea is the main pathogen to cause kiwifruit postharvest soft during fruit senescence. In this study, a comprehensive study that integrates physiological, multi-omic, and microbiome analyses to elucidate a temperature-sensitive feedback loop driving postharvest decay in fruit. Our work identifies a novel mechanism by which a necrotrophic pathogen exploits host-derived sugars to accelerate fruit decaya process effectively mitigated by cold storage. Starch and sucrose metabolism is the main metabolism path way during kiwifruit senescence. Increasing sugar promotes B. dothidea pathogenicity. Meanwhile, low temperature storage 4  inhibit starch degradation to sucrose. The findings not only advance our understanding of host-pathogen interactions in postharvest systems but also highlight sugar metabolism as a central hub regulating fungal dominance and disease progression.