Arabidopsis thaliana
Plants often encounter osmotic stress caused by drought, cold, or salinity and have evolved acquired tolerance systems against more severe stresses. We previously found that acquired osmotolerance induced after salt stress is widespread among Arabidopsis thaliana accessions and identified ACQOS as the responsible gene, causing detrimental autoimmunity via ENHANCED DISEASE SUSCEPTIBILITY1 (EDS1) and PHYTOALEXIN DEFICIENT4 (PAD4) under osmotic stress, thereby reducing osmotolerance. However, it remains unclear how the tolerance is established. To reveal the mechanism underlying the acquired osmotolerance, we here isolated an acquired osmotolerance-defective (aod) 13 mutant by screening 30,000 M2 seedlings obtained from ion beam-mutagenized seeds of Bu-5, an accession with acquired osmotolerance.
sample_name
Bu-5
collection_date
2016-04-07
env_broad_scale
missing
env_local_scale
missing
env_medium
agar medium
estimated_size
120Mb
geo_loc_name
Germany: Burghaun/Rhon)
isol_growth_condt
missing
lat_lon
missing
num_replicons
missing
ploidy
diploid
project_name
Genome sequence of Arabidopsis thaliana Bu-5 wild type and the mutant
propagation
sexual
cultivar
missing
ecotype
missing
isolate
missing
strain
missing
BioSampleModel
MIGS.eu