FoxK1 and FoxK2 cooperate with ORF45 to promote late lytic replication of Kaposi sarcoma-associated herpesvirus
Lytic replication is essential for persistent infection of Kaposi sarcoma-associated herpesvirus (KSHV) and the pathogenesis of related diseases, and many cellular pathways are hijacked by KSHV proteins to initiate and control the lytic replication of this virus. However, the machinery involved in KSHV lytic replication from the early to the late phases remains largely