Protective Effect of Compound K against Podocyte Injury in Chronic Kidney Disease by Maintaining Mitochondrial Homeostasis
Chronic Kidney Disease (CKD) represents a formidable health challenge, frequently culminating in end-stage renal disease with high morbidity and mortality rates. Podocyte injury is identified as a critical factor in the progression, attributable to disruptions in mitochondrial homeostasis and associated cellular dysfunction. Compound K (CK), a metabolite of ginsenosides, demonstrates potential in mitigating renal damage through its protective effects on podocytes.