High-throughput sequencing in ARID1A knockout MDA-MB-231 cells [ATAC-seq]
ARID1A has been suggested as a key regulator for anti-tumor immunity. In the present study, we evaluated the role of ARID1A deficiency in inducing adaptive immune resistance in triple negative breast cancer. To investigate global chromatin remodeling, we performed ATAC–seq in ARID1A knockout and control MDA-MB-231 cells. Overall design: We performed ATAC-seq in ARID1A knockout and control MDA-MB-231 cells.