ALKBH5-mediated m6A modification of 5-HT3A mRNA in primary sensory neurons promotes neuropathic pain behaviors
trigeminal-mediated nerve injury increases the expression of the m6A demethylase ALKBH5 in the injured TG, which elevates the level of 5-HT3A in TG and induces pain-related behaviors. Overall design: transcripts  m6A profiles in rat trigeminal ganglion treated with NC-up (n = 3) or ALKBH5-up  (n = 3)