Dysbiosis-mediated Lactobacillus johnsonii and acetate depletion jointly alters intestinal inflammatory response on the offspring by maternal TDCPP exposure.
Tri (1,3-dichloropropyl) phosphate (TDCPP) and its metabolite BDCPP could induce reproductive toxicity. However, the gut bacteria structure and function after maternal exposure to TDCPP were still rare in pregnant rat model.