The post-transcriptional regulatory protein CsrA amplifies its targetome through direct interactions with stress-response reg-ulatory hubs: the EvgA and AcnA cases.
RNA-sequencing files for Escherichia coli K-12 wild type and two derivatives (acnA genomic CsrA binding sites mutant and evgA genomic CsrA binding sites mutant) grown in biological duplicates up to three different growth conditions: early exponential (OD600~0.8), late exponential (OD600~1.5) and stationary (OD600~2.5).