Atomic Vacancies of Molybdenum Disulfide Nanoparticles Stimulate Mitochondrial Biogenesis
Atomic vacancies rich MoS2 nanoflowers stimulate mitochondrial function by reducing cellualr ROS  generation and  upregulating the expression of genes required for mitochondrial biogenesis. Overall design: Evaluating the effect of atomic vacancies  in MoS2 nanoparticles on hMSC fate. Untreated control hMSCs are compared to hMSCs treated with MoS2 nanoparticles containing low (1:1) or high (1:6) vacancies.