Transcriptional signature for dysplastic epidermal development in human pre-malignant skin
This study aims to identify molecular mechanisms driving the progression of Actinic Keratosis (AK) to Squamous Cell Carcinoma (SCC). The research seeks to explore molecular markers distinguishing normal and dysplastic epidermal regions in AK lesions. Unitmately, develop targeted therapies that provide long-term protection against SCC recurrence.