ATAC-seq data of mouse embryonic stem cells treated with dTAG for SALL4 depletion against untreated control
This study investigates the role of SALL4 in regulating chromatin accessibility. SALL4 was tagged with FKBP to enable dTAG-inducible degradation. ATAC-seq was performed at multiple time-points (1 hour, 4 hours and 8 hours) following dTAG treatment to capture changes in chromatin accessibility upon SALL4 depletion. For each time point, two biological replicates were processed, each with two technical replicates. This dataset aims to characterise the immediate effects of SALL4 loss on chromatin accessibility.