Basal-to-inflammatory transition enhances basal cell carcinoma (BCC) therapy resistance via crosstalk with a pro-inflammatory stromal niche [RNA-seq]
The goal of these RNA-seq experiments was to determine transcriptomic gene expression changes caused by Il1 or Osm (or both) treatments of mouse BCC cells at 24h and 48h timepoints versus PBS control Overall design: 2 biological replicates each