Elucidating the mechanisms underlying H3K18la contributes to NSCLC progression
Histone lactylation has been served as a novel epigenetic modification that directly stimulates gene transcription from chromatin. In order to study the regulatory mechanisms of H3K18la in non-small cell lung cancer (NSCLC), we performed ChIP-Seq to identify its target genes in NCI-H1299 cell lines. Overall design: ChIP-Seq with anti-H3K18la in NCI-H1299 cells