Two unequally redundant 'helper' immune receptor families mediate Arabidopsis intracellular 'sensor' immune receptor functions
Investigate  the function of RNLs in transcriptional reprogramming during Pf0-1-induced PTI and ETI, using time-resolved transcriptomics. To do so we subjected Col-0, adr1 triple, nrg1.1 nrg1.2 and helperless mutants to infections with Pf0-EV (RNL-(in)dependent PTI), Pf0-AvrRps4 (fully RNL-dependent ETI + PTI), Pf0-AvrRpt2 (partial RNL-dependent ETI + PTI) or Pf0-AvrRpm1 (RNL-independent ETI + PTI). Overall design: Time resolved transcriptomics (T0, 0.5h, 4h  and 8h) of 4 Arabidopsis thaliana genotypes (Col-0, nrg1.1 nrg1.2 , adr1 triple and helperless) infiltrated with 4 bacterial treatments (Pf0-EV (RNL-(in)dependent PTI), Pf0-AvrRps4 (fully RNL-dependent ETI + PTI), Pf0-AvrRpt2 (partial RNL-dependent ETI + PTI) or Pf0-AvrRpm1 (RNL-independent ETI + PTI))