Caffeic Acid Alleviates Sepsis-Induced Myocardial Injury Via SIRT7-Regulated H3K14 Lactylation
Sepsis-induced myocardial injury represents a critical clinical condition with limited therapeutic options and high mortality rates. This groundbreaking discovery establishes a novel paradigm wherein CA exerts cardioprotection through dual mechanisms regulating lactylation, providing an innovative epigenetic therapeutic strategy for SIMI treatment based on histone lactylation modulation.