A TLR4/Traf6-dependent signaling pathway mediates NCoR co-activator complex formation for inflammatory gene activation [ATAC]
To investigate the enhancer/promoter activity in TLR4-regulated inflammatory genes, we used bone marrow-derived macrophages treated with or without KLA. We then analyzed open chromatin regions obtained from ATAC-seq data. Overall design: ATAC peak-associated H3K27ac analysis for active enhancer/promoter regions