Neonatal brain injury unravels transcriptional and signaling changes underlying the reactivation of cortical progenitors
We used chronic neonatal hypoxia as a rodent model of early brain injury, to investigate the reactivation of cortical progenitors at postnatal times. We performed gene expression profiling analysis using data obtained from RNA-seq of normoxic and hypoxic brains at embryonic, P2 and P12 time points Overall design: Comparative gene expression profiling analysis of RNA-seq data for progenitors (tdTomato+) cells in normoxic or hypoxic conditions at various time points (E13, E15 and P12)