Contrasting Mutant and Overexpressor Phenotypes Position KIN10 as a Central Growth-Defense Integrator
Plants continuously balance growth and defense in dynamic environments where energy supply, microbial cues, and developmental programs interact intimately. The Sucrose non-fermenting-1 Related Kinase 1 (SnRK1) complex is widely recognized as a central metabolic hub, yet how its catalytic subunit KIN10 coordinates energy status with organ-specific developmental and immune outputs remains poorly understood. Our work fills this knowledge gap by combining transcriptomics, hormone profiling, pathogen assays, and redox phenotyping to elucidate how SnRK1/KIN10 shapes biotic stress responses in both roots and shoots. Our dataset shows how KIN10 orchestrates distinct root vs. shoot transcriptional landscapes and tunes whole-plant immune amplitude and biotic stress outcomes. Overall design: RNAseq profiling of wild-type A.thaliana (Col-0 and Ler-0) and mutants with knock-out (kin10) or overexpression (KIN10-OE) of SnRK1.1/KIN10.