ILF3 mediates lipid metabolism to promote pancreatic cancer development by regulating CPT1A-activated PPARa signaling pathway
Interleukin enhancer binding factor 3 (ILF3) serves as a coactivator for transcription and has been associated with the growth and spread of tumors, despite its unclear involvement in pancreatic cancer (PC). To elucidate the significance of ILF3 in PC, we examined its expression levels at both the tissue and cellular levels.