Stacking the odds: multiple sites for HSV-1 latency
investigated the possible contribution of various cells (i.e., B cells, DCs, fibroblasts, glial cells, ILCs, macrophages, microglia, monocytes, NK cells, neurons, neutrophils, and T cells) isolated from TG of latently infected mice to HSV-1 latency Overall design: immune cells were enriched and pooled back with original TG single cells, so that multiple type of cellls in TG can be analyzed by single cell sequencing