GSE208567
Epigenetically-controlled tumor antigens derived from splice junctions between exons and transposable elements [ChIP-seq]
To analyze regulation of JET expression, we generated Setdb1-deficient MC38 and B16OVA cells, using lentivirus-based CRISPR/Cas9. Ctrl and KO cells lines were used to perform ChIP-seq against H3K9me3 and H3K9Ac, with 3 biological replicates. Overall design: We analyzed H3K9me3 and H3K9Ac signals, in particular on loci involved in forming junctions between exons and TEs.