GSE211217
IFN-g and Androgens Synergistically Disrupt Mitochondrial Function in Murine Myocytes
We report here, aberrant cardiovascular activity in vivo in an autoimmune mouse model with persistent low-level expression of IFN-g (designated ARE mice) linked with androgen exposure, results in a mitochondrial and metabolic deficiency. Overall design: 4 WT and 4 ARE-/- male mice hearts are examined via bulk RNAseqencing