GSE161243
CPT1A Over-expression Increases Reactive Oxygen Species in the Mitochondria and Promotes Antioxidant Defenses in Prostate Cancer
The goal of this study was to identify gene expression changes and pathways associated with CPT1A deficiency ( knockdown, KD) and overexpression (OE) of prostate cancer cells Overall design: RNA-seq was used to define the effects of CPT1A knokdown (KD) or overexpression (OE)  on gene expression in LNCaP-C4-2 castration resistant prostate cancer cells