GSE133182
SME1 deficiency results in oxidative stress tolerance
In this work, we describe a nonsense mutation in SME1 that alleviates photorespiratory cell death in the Arabidopsis thaliana cat2-2 mutant. A SME1 loss-of-function mutant (sme1-1) and wild-type Col-0 were subjected to mRNA-seq for differential gene expression and alternative splicing analysis. This revealed a massive transcript reprogramming and occurrence of splicing defects caused by SME1 deficiency. For more details see the publication. Overall design: 2 genotypes: sme1-1 and wild-type Col-0  X  3 replicates = 6 samples