GSE124005
Genomic instability in the naturally and prematurely aged myocardium [RNA-Seq]
Our data demonstrate that genomic instability does not evidently contribute to naturally aging of the mouse heart and support the contention that the endogenous DNA repair machinery is remarkably active to maintain genomic integrity in cardiac cells throughout life. Overall design: Investigation of variants in RNAseq for four different accelerated aging mouse models as well as natural aging at 3 different time points, each with 4 replicates.