GSE203111
Enhancing GAT-3 in thalamic astrocytes promotes resilience to brain injury in rodents
Thalamic inflammation mediates enhanced seizure risk and altered cognition-related cortical states through astrocytic GAT-3 in mice. Overall design: RNA-sequencing data from ipsilateral (eGFP-pos and eGFP-neg) and contralateral (eGFP-neg) VB thalamic astrocytes three weeks after unilateral injection of AAV2/5-Gfa104-eGFP in VB thalamus in mice.