GSE128511
Loss of Pitx2c predisposes to arrhythmia and atrial fibrillation pathologies via structural and metabolic dysfunction
RNAseq profiling of embryonic zebrafish hearts was performed to identify differentially expressed genes downstream of Pitx2c. Overall design: Embryonic hearts were dissected from wild-type, pitx2c mutants, and pitx2c gain-of-function (OE) embryos at 56 hpf