PRJNA495716
Mus musculus strain:c57BL/6 Genome sequencing
It is known that stress can induce cardiovascular dysfunctions, but the underlying mechanisms are still not well elucidated. The present study aimed to test the hypothesis that alterations of DNA methylation in some cardiac genes may be a mechanism. A mouse model of chronic restraint stress (CRS) was used. Cardiac DNA methylation were sequenced respectively by representation bisulfite sequencing (RRBS).