GSE201924
Genome-wide maps of H3K36me3 ChIP-seq state in lung smooth muscle cells
To examine the effects of Tet3-inactivation on genome-wide transcriptional fidelity, we determined the binding profiles of H3K36me3, a specific form of H3K36me3 that marks transcriptional elongation sites, by ChIP-seq analysis. Overall design: Examination of control and Tet3-inactivation smooth muscle cells.