GSE201560
GS ablation leads to elevated mTORC1 activation in mouse livers harboring c-Met/?N90-ß-catenin
Hepatic GS loss leads to increased mTORC1 signaling and ribosome biogenesis in response to ß-catenin activation Overall design: Seven-week-old GS WT and KO male mice were injected with vehicle or c-Met/?N90-ß-catenin/SB10 plasmids, and livers were harvested 0, 2 or 3 weeks after the injection for RNA-sequencing analysis