ena-STUDY-HZI - HELMHOLTZ CENTRE FOR INFECTION RESEARCH, BRAUNSCHWEIG, GERMANY-03-09-2014-08:47:09:820-204
Analysis of the CD94 encoding gene Klrd1 revealed isogenic DBA/2 mice
DBA/2J mice lack CD94 expression due to a deletion in the Klrd1 gene that occurred in the 1980s. Depending on the breeder we identified two DBA/2 variants with and without the deletion representing the old and new genotype that is still reflected in different batches of BXD lines and being a useful tool to study the role of CD94.
DBA/2J mice lack CD94 expression due to a deletion in the Klrd1 gene that occurred in the 1980s. Depending on the breeder we identified two DBA/2 variants with and without the deletion representing the old and new genotype that is still reflected in different batches of BXD lines and being a useful tool to study the role of CD94.