GSE202288
Cell State Dependent Alteration of Melanoma Plasticity and Immunity by the Circadian Transcription Factor Bmal1 [ChIP-Seq]
The circadian transcription factor Bmal1 accelerates melanoma tumorigenesis through initial cell state dependent mechanisms in which Hif1??, Sox9 or Myh9, MRTF-SRF, AP-1 are involved and modulate tumor microenvironment to favor tumor growth Overall design: CHIP-seq of H3K4me3 and H3K27me3 in cells of different Bmal1 status