GSE163318
Genome-wide ChIP-seq analysis of H2A.Z and H2A.Zac in HeLa Kyoto cells upon knockdown of JAZF1
The goal was to investigate genome-wide if JAZF1 influences the deposition as well as the acetylation of the histone variant H2A.Z. ChIP-seq analysis revealed that depletion of JAZF1 leads to reduced acetylation of H2A.Z at specific sites. Overall design: Genome-wide H2A.Z and H2A.Zac profiles of HeLa Kyoto cells upon knockdown of JAZF1 compared to control treatment (with biological replicates) were generated by ChIP-sequencing