GSE161754
Shutoff of host transcription triggers a toxin-antitoxin system to cleave phage RNA and abort infection [toxIN_riftreatment_RNAseq]
The purpose of this experiment was to characterize ToxN activity following activation by rifampicin treatment. Overall design: Two replicates each of +toxIN (pBR322-toxIN) and -toxIN (pBR322 empty vector) cells 0,2.5,5,10,20, and 30 min post-rifampicin treatment.